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A Molecular Genetic Model of Astrocytoma
Histopathology. D.N. Louis. As the molecular events
responsible for astrocytoma formation and progression are
being clarified, it is becoming possible to correlate these
alterations with the specific histopathological and biological
features of astrocytoma, anaplastic astrocytoma and
glioblastoma multiforme. In WHO grade II astrocytomas,
autocrine stimulation by the platelet-derived growth factor
system coupled with inactivation of the p53 gene may lead to
a growth stimulus in the face of decreased cell death with
slow net growth ensuing. Such cells would also have defective
responses to DNA damage and impaired DNA repair, setting
the stage for future malignant change.
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